File:Mouse E18 neurovasculature MicroCT.jpg

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Mouse E18 Cerebral Vasculature MicroCT

Complement activation, in particular C5a, may contribute to neuropathology and adverse outcomes during malaria in pregnancy. Animal model results show that experimental malaria in pregnancy nduces neurocognitive deficits in offspring and suggest novel targets for intervention.

Links: Mouse E18 Cerebral Vasculature MicroCT | Abnormal Development - Malaria | Computed Tomography | Developmental Origins of Health and Disease | Mouse Development

Reference

McDonald CR, Cahill LS, Ho KT, Yang J, Kim H, Silver KL, Ward PA, Mount HT, Liles WC, Sled JG & Kain KC. (2015). Experimental Malaria in Pregnancy Induces Neurocognitive Injury in Uninfected Offspring via a C5a-C5a Receptor Dependent Pathway. PLoS Pathog. , 11, e1005140. PMID: 26402732 DOI.

Copyright

© 2015 McDonald et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited

Original image adjusted in size and labelling.


Cite this page: Hill, M.A. (2024, April 25) Embryology Mouse E18 neurovasculature MicroCT.jpg. Retrieved from https://embryology.med.unsw.edu.au/embryology/index.php/File:Mouse_E18_neurovasculature_MicroCT.jpg

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© Dr Mark Hill 2024, UNSW Embryology ISBN: 978 0 7334 2609 4 - UNSW CRICOS Provider Code No. 00098G

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current11:51, 28 September 2015Thumbnail for version as of 11:51, 28 September 2015960 × 364 (68 KB)Z8600021 (talk | contribs)==Mouse E18 Cerebral Vasculature MicroCT== Complement activation, in particular C5a, may contribute to neuropathology and adverse outcomes during malaria in pregnancy. Animal model results show that experimental malaria in pregnancy nduces neurocogniti...

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