Template:Genital terms: Difference between revisions

From Embryology
mNo edit summary
mNo edit summary
Line 1: Line 1:
{| class="wikitable mw-collapsible mw-collapsed"
{| class="wikitable mw-collapsible mw-collapsed"
! [[Genital System Development|Genital System]] Terms (expand to view) 
! [[Genital System Development|Genital System Terms]] (expand to view) 
|-
|-
| Note there are additional glossaries associated with [[Template:Spermatozoa Terms|spermatozoa]], [[Template:OocyteTerms|oocyte]] [[Template:Renal terms|renal]].
| Note there are additional glossaries associated with [[Template:Spermatozoa Terms|spermatozoa]], [[Template:OocyteTerms|oocyte]] [[Template:Renal terms|renal]].


* '''androgens''' -  male sex hormones, such as testosterone, or the more potent dihydrotestosterone (DHT, 5α-dihydrotestosterone, androstanolone, 5α-androstan-17β-ol-3-one), formed by the enzyme 5α-reductase. Dihydrotestosterone).
* '''anti-mullerian hormone''' (AMH, Mullerian Inhibiting Substance, MIS) A secreted glycoprotein factor (transforming growth factor-beta, TGF-beta superfamily) that regulates gonadal and genital tract development. In the male embryo, the Sertoli cell secrete AMH and inhibit paramesonephric (Mullerian) duct development. In postnatal males, AMH increases during the first month, reaching peak level at 6 months of age, and then slowly declines during childhood falling to low levels in puberty. In reproductive age women, AMH is produced in the ovary by the granulosa cells surrounding preantral and small antral follicles and serum levels may reflect the remaining follicle cohort and decrease with age.
* '''Müllerian inhibiting substance''' - (MIS, anti-Müllerian hormone, AMH) A secreted glycoprotein factor (transforming growth factor-beta, TGF-beta superfamily) that regulates gonadal and genital tract development. In the male embryo, the Sertoli cell secrete AMH and inhibit paramesonephric (Mullerian) duct development. In postnatal males, AMH increases during the first month, reaching peak level at 6 months of age, and then slowly declines during childhood falling to low levels in puberty. In reproductive age women, AMH is produced in the ovary by the granulosa cells surrounding preantral and small antral follicles and serum levels may reflect the remaining follicle cohort and decrease with age.


human Müllerian inhibiting substance (MIS)/anti-Müllerian hormone (AMH) gene has 5 exons and 4 introns and encodes a 70 kDa monomer with two N-linked glycosylation sites (circles). The disulfide-linked homodimer is activated by biosynthetic proteases to produce the 25-kDa carboxy-terminal dimer. This domain is responsible for its bioactivity.
|}
|}
{{Terms lists}}<noinclude>[[Category:Genital]][[Category:Terms]][[Category:Glossary]][[Category:Template]]</noinclude>
{{Terms lists}}<noinclude>[[Category:Genital]][[Category:Terms]][[Category:Glossary]][[Category:Template]]</noinclude>

Revision as of 11:16, 1 June 2017

Genital System Terms (expand to view) 
Note there are additional glossaries associated with spermatozoa, oocyte renal.
  • androgens - male sex hormones, such as testosterone, or the more potent dihydrotestosterone (DHT, 5α-dihydrotestosterone, androstanolone, 5α-androstan-17β-ol-3-one), formed by the enzyme 5α-reductase. Dihydrotestosterone).
  • anti-mullerian hormone (AMH, Mullerian Inhibiting Substance, MIS) A secreted glycoprotein factor (transforming growth factor-beta, TGF-beta superfamily) that regulates gonadal and genital tract development. In the male embryo, the Sertoli cell secrete AMH and inhibit paramesonephric (Mullerian) duct development. In postnatal males, AMH increases during the first month, reaching peak level at 6 months of age, and then slowly declines during childhood falling to low levels in puberty. In reproductive age women, AMH is produced in the ovary by the granulosa cells surrounding preantral and small antral follicles and serum levels may reflect the remaining follicle cohort and decrease with age.
  • Müllerian inhibiting substance - (MIS, anti-Müllerian hormone, AMH) A secreted glycoprotein factor (transforming growth factor-beta, TGF-beta superfamily) that regulates gonadal and genital tract development. In the male embryo, the Sertoli cell secrete AMH and inhibit paramesonephric (Mullerian) duct development. In postnatal males, AMH increases during the first month, reaching peak level at 6 months of age, and then slowly declines during childhood falling to low levels in puberty. In reproductive age women, AMH is produced in the ovary by the granulosa cells surrounding preantral and small antral follicles and serum levels may reflect the remaining follicle cohort and decrease with age.
Other Terms Lists  
Terms Lists: ART | Birth | Bone | Cardiovascular | Cell Division | Endocrine | Gastrointestinal | Genital | Genetic | Head | Hearing | Heart | Immune | Integumentary | Neonatal | Neural | Oocyte | Palate | Placenta | Radiation | Renal | Respiratory | Spermatozoa | Statistics | Tooth | Ultrasound | Vision | Historic | Drugs | Glossary