File:Duodenal atresia 01.jpg
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Duodenal Atresia
Upper gastrointestinal series showing complete obstruction to the flow of contrast at the second portion of the duodenum. There is also contrast filling of the biliary tree above the duodenal bulb noted (arrow).
Duodenal atresia (DA) occurs in approximately 1 in 2500–7500 live births without a sex-associated difference. Approximately 25–40% of infants with duodenal atresia have Trisomy 21. Approximately 8% of infants with Down syndrome have duodenal atresia. There is also an association of VACTERL anomalies (vertebral, anorectal, cardiac, tracheoesophageal, renal, and limb anomalies).
ICD-11 LB14 Structural developmental anomalies of duodenum - Any congenital defect of duodenum that results from interference with the normal growth and differentiation of the fetus. Such defects can arise at any stage of embryonic development, vary greatly in type and severity, and are caused by a wide variety of determining factors, including genetic mutations, chromosomal aberrations, teratogenic agents, and environmental factors. Most developmental defects are apparent at birth, especially any structural malformation, but some becomes evident later. |
- Links: duodenal atresia | gastrointestinal abnormalities | image 1 | image 2
Reference
Puvabanditsin S, Botwinick M, Chen CW, Joshi A & Mehta R. (2018). Biliary Tract Abnormalities as a Cause of Distal Bowel Gas in Neonatal Duodenal Atresia. Case Rep Surg , 2018, 8041427. PMID: 30046504 DOI.
Copyright
© 2018 Surasak Puvabanditsin et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
CRIS2018-8041427.002.jpg
Cite this page: Hill, M.A. (2024, April 27) Embryology Duodenal atresia 01.jpg. Retrieved from https://embryology.med.unsw.edu.au/embryology/index.php/File:Duodenal_atresia_01.jpg
- © Dr Mark Hill 2024, UNSW Embryology ISBN: 978 0 7334 2609 4 - UNSW CRICOS Provider Code No. 00098G
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