Talk:Abnormal Development - Zika Virus: Difference between revisions

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==2016==
==2016==


* Pyriproxyfen is a larvicide (manufactured by Sumitomo Chemical) was used in a massive government-run program tasked to control the mosquito population in the country. http://www.techtimes.com/articles/133548/20160214/monsanto-larvicide-not-zika-virus-true-cause-of-brazils-microcephaly-outbreak-doctors.htm
* Pyriproxyfen is a larvicide (manufactured by Sumitomo Chemical) was used in a massive government-run program tasked to control the mosquito population in the country. http://www.techtimes.com/articles/133548/20160214/monsanto-larvicide-not-zika-virus-true-cause-of-brazils-microcephaly-outbreak-doctors.htm PMID 23843982





Revision as of 09:58, 21 February 2016

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Cite this page: Hill, M.A. (2024, April 19) Embryology Abnormal Development - Zika Virus. Retrieved from https://embryology.med.unsw.edu.au/embryology/index.php/Talk:Abnormal_Development_-_Zika_Virus


2016


2015

Zika virus in Brazil and the danger of infestation by Aedes (Stegomyia) mosquitoes

Rev Soc Bras Med Trop. 2015 Dec 22. pii: S0037-86822015005003102. [Epub ahead of print]

Marcondes CB1, Ximenes MF2.

Abstract

Zika virus, already widely distributed in Africa and Asia, was recently reported in two Northeastern Brazilian: State of Bahia and State of Rio Grande do Norte, and one Southeastern: State of São Paulo. This finding adds a potentially noxious virus to a list of several other viruses that are widely transmitted by Aedes (Stegomyia) aegypti and Aedes (Stegomyia) albopictus in Brazil. The pathology and epidemiology, including the distribution and vectors associated with Zika virus, are reviewed. This review is focused on viruses transmitted by Aedes (Stegomyia) mosquitoes, including dengue, Chikungunya, Zika, Mayaro, and yellow fever virus, to emphasize the risks of occurrence for these arboviruses in Brazil and neighboring countries. Other species of Aedes (Stegomyia) are discussed, emphasizing their involvement in arbovirus transmission and the possibility of adaptation to environments modified by human activities and introduction in Brazil.

PMID 26689277

Zika virus and the never-ending story of emerging pathogens and transfusion medicine

Blood Transfus. 2015 Nov 5:1-6. doi: 10.2450/2015.0066-15. [Epub ahead of print]

Marano G1, Pupella S1, Vaglio S1,2, Liumbruno GM1, Grazzini G1.

Abstract

In the last few years, the transfusion medicine community has been paying special attention to emerging vector-borne diseases transmitted by arboviruses. Zika virus is the latest of these pathogens and is responsible for major outbreaks in Africa, Asia and, more recently, in previously infection-naïve territories of the Pacific area. Many issues regarding this emerging pathogen remain unclear and require further investigation. National health authorities have adopted different prevention strategies. The aim of this review article is to discuss the currently available, though limited, information and the potential impact of this virus on transfusion medicine. PMID 26674815

2014

Stress responses in flavivirus-infected cells: activation of unfolded protein response and autophagy

Front Microbiol. 2014 Jun 3;5:266. doi: 10.3389/fmicb.2014.00266. eCollection 2014.

Blázquez AB1, Escribano-Romero E1, Merino-Ramos T1, Saiz JC1, Martín-Acebes MA2.

Abstract

The Flavivirus is a genus of RNA viruses that includes multiple long known human, animal, and zoonotic pathogens such as Dengue virus, yellow fever virus, West Nile virus, or Japanese encephalitis virus, as well as other less known viruses that represent potential threats for human and animal health such as Usutu or Zika viruses. Flavivirus replication is based on endoplasmic reticulum-derived structures. Membrane remodeling and accumulation of viral factors induce endoplasmic reticulum stress that results in activation of a cellular signaling response termed unfolded protein response (UPR), which can be modulated by the viruses for their own benefit. Concomitant with the activation of the UPR, an upregulation of the autophagic pathway in cells infected with different flaviviruses has also been described. This review addresses the current knowledge of the relationship between endoplasmic reticulum stress, UPR, and autophagy in flavivirus-infected cells and the growing evidences for an involvement of these cellular pathways in the replication and pathogenesis of these viruses. KEYWORDS: West Nile virus; autophagy; dengue virus; endoplasmic reticulum stress; flavivirus; unfolded protein response; virus replication

PMID 24917859