Talk:Urinary Bladder Development - Revision history

Z8600021 at 07:53, 7 December 2021

2021-12-07T07:53:06Z

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 {{Talk Page}}
 ===Immunohistochemical expression analysis of the human fetal lower urogenital tract===
 {{#pmid:30287094}}

Z8600021 at 23:43, 3 Mayıs 2020

2020-05-03T23:43:46Z

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 PMCID: PMC5163740
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Z8600021 at 23:38, 3 Mayıs 2020

2020-05-03T23:38:42Z

← Older revision		Revision as of 09:38, 4 Mayıs 2020
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	==Historic==		==Historic==

			{{Ref-Vermooten1951}}

	Congenital Cystic Dilatation of the Renal Collecting Tubules: A New Disease Entity		Congenital Cystic Dilatation of the Renal Collecting Tubules: A New Disease Entity
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	Yale J Biol Med. 1951 Jun; 23(6): 450–453.		Yale J Biol Med. 1951 Jun; 23(6): 450–453.
	PMCID: PMC2599097		PMCID: PMC2599097

			{{Ref-SmithStrasberg1946}}

	CYSTS OF THE GENITAL DUCTS, MÜLLERIAN AND WOLFFIAN		CYSTS OF THE GENITAL DUCTS, MÜLLERIAN AND WOLFFIAN
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	PMCID: PMC1582939		PMCID: PMC1582939

			{{Ref-RuscheBacon1939}}

	Congenital Renal Anomalies: With Special Reference to Horseshoe Kidney		Congenital Renal Anomalies: With Special Reference to Horseshoe Kidney
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	PMCID: PMC1660477		PMCID: PMC1660477
	{{#pmid:18745137}}		{{#pmid:18745137}}

			{{Ref-Randall1937}}

	THE ORIGIN AND GROWTH OF RENAL CALCULI		THE ORIGIN AND GROWTH OF RENAL CALCULI
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	Ann Surg. 1937 Jun; 105(6): 1009–1027. doi: 10.1097/00000658-193706000-00014		Ann Surg. 1937 Jun; 105(6): 1009–1027. doi: 10.1097/00000658-193706000-00014
	PMCID: PMC1390483		PMCID: PMC1390483

			{{Ref-MortonJones1936}}

	OBSTRUCTIONS ABOUT THE MESENTERY IN INFANTS		OBSTRUCTIONS ABOUT THE MESENTERY IN INFANTS
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	PMCID: PMC1390255		PMCID: PMC1390255
	{{#pmid:17856875}}		{{#pmid:17856875}}

			{{Ref-Bell1935}}

	Cystic Disease of the Kidneys		Cystic Disease of the Kidneys
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	Am J Pathol. 1935 May; 11(3): 373–418.5.		Am J Pathol. 1935 May; 11(3): 373–418.5.
	PMCID: PMC1910969		PMCID: PMC1910969

			{{Ref-Oppenheimer1934}}

	Polycystic Disease of the Kidney		Polycystic Disease of the Kidney
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	PMCID: PMC5163740		PMCID: PMC5163740

	{{Ref-Abbott1935}		{{Ref-Abbott1935}}

	ANOMALIES OF THE GENITO-URINARY TRACT		ANOMALIES OF THE GENITO-URINARY TRACT

Z8600021 at 02:49, 3 Mayıs 2020

2020-05-03T02:49:51Z

← Older revision		Revision as of 12:49, 3 Mayıs 2020
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	Ind Med Gaz. 1933 Sep; 68(9): 513–515.		Ind Med Gaz. 1933 Sep; 68(9): 513–515.
	PMCID: PMC5163740		PMCID: PMC5163740

			{{Ref-Abbott1935}

	ANOMALIES OF THE GENITO-URINARY TRACT		ANOMALIES OF THE GENITO-URINARY TRACT
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	PMCID: PMC382003		PMCID: PMC382003
	{{#pmid:20317935}}		{{#pmid:20317935}}

			{{Ref-PapinEisendrath1927}}

	CLASSIFICATION OF RENAL AND URETERAL ANOMALIES		CLASSIFICATION OF RENAL AND URETERAL ANOMALIES
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	PMCID: PMC1399333		PMCID: PMC1399333

			{{Ref-Piersol1927}}

	Polycystic Disease of the Kidney		Polycystic Disease of the Kidney
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	{{#pmid:21408945}}		{{#pmid:21408945}}


			{{Ref-HinmanGibsonKutzmann1924}}

	CYSTS OF THE WOLFFIAN BODY		CYSTS OF THE WOLFFIAN BODY
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	Ann Surg. 1924 May; 79(5): 762–769. doi: 10.1097/00000658-192405000-00012		Ann Surg. 1924 May; 79(5): 762–769. doi: 10.1097/00000658-192405000-00012
	PMCID: PMC1399572		PMCID: PMC1399572

			{{Ref-Eisendrath1917}}

	CONGENITAL STRICTURES AND SPIRAL TWISTS OF THE URETERS		CONGENITAL STRICTURES AND SPIRAL TWISTS OF THE URETERS
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	Ann Surg. 1917 May; 65(5): 552–559.		Ann Surg. 1917 May; 65(5): 552–559.
	PMCID: PMC1426593		PMCID: PMC1426593

			{{Ref-Braasch1912}}

	VIII. The Clinical Diagnosis of Congenital Anomaly in the Kidney and Ureter		VIII. The Clinical Diagnosis of Congenital Anomaly in the Kidney and Ureter
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	PMCID: PMC1407353		PMCID: PMC1407353
	{{#pmid:17862922}}		{{#pmid:17862922}}

			{{Ref-Eisendrath1912}}

	CONGENITAL MALFORMATIONS OF THE URETERS		CONGENITAL MALFORMATIONS OF THE URETERS
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	Ann Surg. 1912 Apr; 55(4): 571–592. doi: 10.1097/00000658-191204000-00007		Ann Surg. 1912 Apr; 55(4): 571–592. doi: 10.1097/00000658-191204000-00007
	PMCID: PMC1407239		PMCID: PMC1407239

			{{Ref-Spicer1909}}

	Dystocia due to Distension of the Urinary Bladder of the Fœtus, with Remarks on Renal Secretion in utero		Dystocia due to Distension of the Urinary Bladder of the Fœtus, with Remarks on Renal Secretion in utero
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	PMCID: PMC2046649		PMCID: PMC2046649
	SummaryPage BrowsePDF–3.2MCitation		SummaryPage BrowsePDF–3.2MCitation

			{{Ref-Hamann1902}}

	Spindle-Shaped Dilatations and Tortuosity of the Ureters in the Fetus		Spindle-Shaped Dilatations and Tortuosity of the Ureters in the Fetus

Z8600021 at 02:44, 3 Mayıs 2020

2020-05-03T02:44:52Z

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 ==2018==
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 A defect of development, in which the bladder is implicated, is known under the name of extroversion of the bladder. In this condition the lower part of the abdominal wall and the anterior wall of the bladder are wanting, so that the fundus of the bladder presents on the abdominal surface, and is pushed forward by the pressure of the viscera within the abdomen, forming a red vascular tumor on which the openings of the ureters are visible. The penis, except the glans, is rudimentary and is cleft on its dorsal surface, exposing the floor of the urethra, a condition known as epispadias. The pelvic bones are also arrested in development.

Z8600021 at 04:04, 29 Ocak 2019

2019-01-29T04:04:23Z

← Older revision		Revision as of 14:04, 29 Ocak 2019
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	{{Template:Talk Page}}		{{Template:Talk Page}}
	==~~10 Most Recent~~==		==2018==
	~~{{10 Most Recent}}~~
	===~~Urinary Bladder Development~~===
	~~<pubmed limit=~~5~~>Urinary Bladder Development<~~/~~pubmed>~~		===Immunohistochemical expression analysis of the human fetal lower urogenital tract===
			{{#pmid:30287094}}

			Differentiation. 2018 Sep - Oct;103:100-119. doi: 10.1016/j.diff.2018.09.004. Epub 2018 Sep 19.

			Shen J1, Isaacson D1, Cao M1, Sinclair A1, Cunha GR1, Baskin L2.

			Abstract
			We have studied the ontogeny of the developing human male and female urogenital tracts from 9 weeks (indifferent stage) to 16 weeks (advanced sex differentiation) of gestation by immunohistochemistry on mid-sagittal sections. Sixteen human fetal pelvises were serial sectioned in the sagittal plane and stained with antibodies to epithelial, muscle, nerve, proliferation and hormone receptor markers. Key findings are: (1) The corpus cavernosum in males and females extends into the glans penis and clitoris, respectively, during the ambisexual stage (9 weeks) and thus appears to be an androgen-independent event. (2) The entire human male (and female) urethra is endodermal in origin based on the presence of FOXA1, KRT 7, uroplakin, and the absence of KRT10 staining. The endoderm of the urethra interfaces with ectodermal epidermis at the site of the urethral meatus. (3) The surface epithelium of the verumontanum is endodermal in origin (FOXA1-positive) with a possible contribution of Pax2-positive epithelial cells implying additional input from the Wolffian duct epithelium. (4) Prostatic ducts arise from the endodermal (FOXA1-positive) urogenital sinus epithelium near the verumontanum. (5) Immunohistochemical staining of mid-sagittal and para-sagittal sections revealed the external anal sphincter, levator ani, bulbospongiosus muscle and the anatomic relationships between these developing skeletal muscles and organs of the male and female reproductive tracts. Future studies of normal human developmental anatomy will lay the foundation for understanding congenital anomalies of the lower urogenital tract.
			Copyright © 2018 International Society of Differentiation. Published by Elsevier B.V. All rights reserved.
			KEYWORDS:
			Human fetal lower urogenital tract; Immunohistochemical; Sagittal sections
			PMID: 30287094 DOI: 10.1016/j.diff.2018.09.004

	==2016==		==2016==

Z8600021 at 05:01, 14 November 2018

2018-11-14T05:01:00Z

← Older revision		Revision as of 15:01, 14 November 2018
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	http://www.jurology.com/article/S0022-5347(09)02320-9/abstract		http://www.jurology.com/article/S0022-5347(09)02320-9/abstract

			==2009==

			===The exstrophy-epispadias complex===

			Orphanet J Rare Dis. 2009 Oct 30;4:23. doi: 10.1186/1750-1172-4-23.

			Ebert AK1, Reutter H, Ludwig M, Rösch WH.
			Author information
			Abstract
			Exstrophy-epispadias complex (EEC) represents a spectrum of genitourinary malformations ranging in severity from epispadias (E) to classical bladder exstrophy (CEB) and exstrophy of the cloaca (EC). Depending on severity, EEC may involve the urinary system, musculoskeletal system, pelvis, pelvic floor, abdominal wall, genitalia, and sometimes the spine and anus. Prevalence at birth for the whole spectrum is reported at 1/10,000, ranging from 1/30,000 for CEB to 1/200,000 for EC, with an overall greater proportion of affected males. EEC is characterized by a visible defect of the lower abdominal wall, either with an evaginated bladder plate (CEB), or with an open urethral plate in males or a cleft in females (E). In CE, two exstrophied hemibladders, as well as omphalocele, an imperforate anus and spinal defects, can be seen after birth. EEC results from mechanical disruption or enlargement of the cloacal membrane; the timing of the rupture determines the severity of the malformation. The underlying cause remains unknown: both genetic and environmental factors are likely to play a role in the etiology of EEC. Diagnosis at birth is made on the basis of the clinical presentation but EEC may be detected prenatally by ultrasound from repeated non-visualization of a normally filled fetal bladder. Counseling should be provided to parents but, due to a favorable outcome, termination of the pregnancy is no longer recommended. Management is primarily surgical, with the main aims of obtaining secure abdominal wall closure, achieving urinary continence with preservation of renal function, and, finally, adequate cosmetic and functional genital reconstruction. Several methods for bladder reconstruction with creation of an outlet resistance during the newborn period are favored worldwide. Removal of the bladder template with complete urinary diversion to a rectal reservoir can be an alternative. After reconstructive surgery of the bladder, continence rates of about 80% are expected during childhood. Additional surgery might be needed to optimize bladder storage and emptying function. In cases of final reconstruction failure, urinary diversion should be undertaken. In puberty, genital and reproductive function are important issues. Psychosocial and psychosexual outcome depend on long-term multidisciplinary care to facilitate an adequate quality of life.
			PMID: 19878548 PMCID: PMC2777855 DOI: 10.1186/1750-1172-4-23


	==2007==		==2007==

Z8600021 at 06:01, 9 March 2017

2017-03-09T06:01:01Z

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 <pubmed limit=5>Urinary Bladder Development</pubmed>
-==2015==
+==2016==
 ==2014==
 ===Morphology of the fetal bladder during the second trimester: Comparing genders===

Z8600021 at 03:42, 31 Mayıs 2016

2016-05-31T03:42:30Z

← Older revision		Revision as of 13:42, 31 Mayıs 2016
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	http://www.jurology.com/article/S0022-5347(09)02320-9/abstract		http://www.jurology.com/article/S0022-5347(09)02320-9/abstract

			==2007==

			===The development of the bladder trigone, the center of the anti-reflux mechanism===

			Development. 2007 Oct;134(20):3763-9. Epub 2007 Sep 19.

			Viana R1, Batourina E, Huang H, Dressler GR, Kobayashi A, Behringer RR, Shapiro E, Hensle T, Lambert S, Mendelsohn C.

			Abstract

			The urinary tract is an outflow system that conducts urine from the kidneys to the bladder via the ureters that propel urine to the bladder via peristalsis. Once in the bladder, the ureteral valve, a mechanism that is not well understood, prevents backflow of urine to the kidney that can cause severe damage and induce end-stage renal disease. The upper and lower urinary tract compartments form independently, connecting at mid-gestation when the ureters move from their primary insertion site in the Wolffian ducts to the trigone, a muscular structure comprising the bladder floor just above the urethra. Precise connections between the ureters and the trigone are crucial for proper function of the ureteral valve mechanism; however, the developmental events underlying these connections and trigone formation are not well understood. According to established models, the trigone develops independently of the bladder, from the ureters, Wolffian ducts or a combination of both; however, these models have not been tested experimentally. Using the Cre-lox recombination system in lineage studies in mice, we find, unexpectedly, that the trigone is formed mostly from bladder smooth muscle with a more minor contribution from the ureter, and that trigone formation depends at least in part on intercalation of ureteral and bladder muscle. These studies suggest that urinary tract development occurs differently than previously thought, providing new insights into the mechanisms underlying normal and abnormal development.
			PMID 17881488

Z8600021 at 01:57, 21 September 2015

2015-09-21T01:57:42Z

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 ==2014==
 ===Morphology of the fetal bladder during the second trimester: Comparing genders===
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 PMID 25434295
 ==2013==