File:Steroid hormone receptor signaling.jpg - Revision history

Z8600021: /* Steroid hormone receptor signaling */

2013-05-25T01:45:49Z

Steroid hormone receptor signaling

← Older revision		Revision as of 12:45, 25 Mayıs 2013
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	~~Original file name~~: ~~Figure 2 Big_nrs05003.f2.gif http~~:~~//www.nursa.org/ejournal/published/05003/big_nrs05003.f2.gif~~		:'''Links:''' [[Endocrine System Development]]

	===Reference===		===Reference===

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	====Copyright====		====Copyright====
	© 2007, Griekspoor et al. This is an open-access article distributed under the terms of the Creative Commons Non-Commercial Attribution License, which permits unrestricted non-commercial use distribution and reproduction in any medium, provided the original work is properly cited.		© 2007, Griekspoor et al. This is an open-access article distributed under the terms of the Creative Commons Non-Commercial Attribution License, which permits unrestricted non-commercial use distribution and reproduction in any medium, provided the original work is properly cited.


			Original file name: Figure 2 Big_nrs05003.f2.gif http://www.nursa.org/ejournal/published/05003/big_nrs05003.f2.gif

	[[Category:Molecular]] [[Category:Endocrine]] [[Category:Cartoon]]		[[Category:Molecular]] [[Category:Endocrine]] [[Category:Cartoon]]

Z8600021: /* Steroid hormone receptor signaling */

2013-05-25T01:45:09Z

Steroid hormone receptor signaling

← Older revision		Revision as of 12:45, 25 Mayıs 2013
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	Copyright © 2007, Griekspoor et al. This is an open-access article distributed under the terms of the Creative Commons Non-Commercial Attribution License, which permits unrestricted non-commercial use distribution and reproduction in any medium, provided the original work is properly cited.		====Copyright====
			© 2007, Griekspoor et al. This is an open-access article distributed under the terms of the Creative Commons Non-Commercial Attribution License, which permits unrestricted non-commercial use distribution and reproduction in any medium, provided the original work is properly cited.

	[[Category:Molecular]] [[Category:Endocrine]] [[Category:Cartoon]]		[[Category:Molecular]] [[Category:Endocrine]] [[Category:Cartoon]]

S8600021: /* Steroid hormone receptor signaling */

2011-09-25T20:50:24Z

Steroid hormone receptor signaling

← Older revision		Revision as of 07:50, 26 September 2011
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	==Steroid hormone receptor signaling==		==Steroid hormone receptor signaling==
	{\|		{\|
	\| width="400px" valign="top"\|Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors. Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock proteins, and translocates to the nucleus. There, the receptor dimerizes, binds specific sequences in the DNA, called Hormone Responsive Elements or HREs, and recruits a number of coregulators that facilitate gene transcription. This latter step can be modulated by receptor antagonists like tamoxifen (T), and cellular signalling pathways.		\| width="400px" valign="top"\|Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors.

			Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock proteins, and translocates to the nucleus.

			There, the receptor dimerizes, binds specific sequences in the DNA, called Hormone Responsive Elements or HREs, and recruits a number of coregulators that facilitate gene transcription.

			This latter step can be modulated by receptor antagonists like tamoxifen (T), and cellular signalling pathways.
	\| '''Legend'''		\| '''Legend'''

S8600021: /* Steroid hormone receptor signaling */

2011-09-25T20:49:50Z

Steroid hormone receptor signaling

← Older revision		Revision as of 07:49, 26 September 2011
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	==Steroid hormone receptor signaling==		==Steroid hormone receptor signaling==
	{\|		{\|
	\| width="400px"\|Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors. Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock proteins, and translocates to the nucleus. There, the receptor dimerizes, binds specific sequences in the DNA, called Hormone Responsive Elements or HREs, and recruits a number of coregulators that facilitate gene transcription. This latter step can be modulated by receptor antagonists like tamoxifen (T), and cellular signalling pathways.		\| width="400px" valign="top"\|Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors. Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock proteins, and translocates to the nucleus. There, the receptor dimerizes, binds specific sequences in the DNA, called Hormone Responsive Elements or HREs, and recruits a number of coregulators that facilitate gene transcription. This latter step can be modulated by receptor antagonists like tamoxifen (T), and cellular signalling pathways.
	\| '''Legend'''		\| '''Legend'''

S8600021: /* Steroid hormone receptor signaling */

2011-09-25T20:49:12Z

Steroid hormone receptor signaling

← Older revision		Revision as of 07:49, 26 September 2011
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	==Steroid hormone receptor signaling==		==Steroid hormone receptor signaling==
	{\|		{\|
	\| Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors. Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock proteins, and translocates to the nucleus. There, the receptor dimerizes, binds specific sequences in the DNA, called Hormone Responsive Elements or HREs, and recruits a number of coregulators that facilitate gene transcription. This latter step can be modulated by receptor antagonists like tamoxifen (T), and cellular signalling pathways.		\| width="400px"\|Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors. Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock proteins, and translocates to the nucleus. There, the receptor dimerizes, binds specific sequences in the DNA, called Hormone Responsive Elements or HREs, and recruits a number of coregulators that facilitate gene transcription. This latter step can be modulated by receptor antagonists like tamoxifen (T), and cellular signalling pathways.
	\| '''Legend'''		\| '''Legend'''

S8600021: /* Steroid hormone receptor signaling */

2011-09-25T20:48:46Z

Steroid hormone receptor signaling

← Older revision		Revision as of 07:48, 26 September 2011
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	==Steroid hormone receptor signaling==		==Steroid hormone receptor signaling==
	Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors. Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock proteins, and translocates to the nucleus. There, the receptor dimerizes, binds specific sequences in the DNA, called Hormone Responsive Elements or HREs, and recruits a number of coregulators that facilitate gene transcription. This latter step can be modulated by receptor antagonists like tamoxifen (T), and cellular signalling pathways.		{\|
			\| Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors. Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock proteins, and translocates to the nucleus. There, the receptor dimerizes, binds specific sequences in the DNA, called Hormone Responsive Elements or HREs, and recruits a number of coregulators that facilitate gene transcription. This latter step can be modulated by receptor antagonists like tamoxifen (T), and cellular signalling pathways.
			\| '''Legend'''
	'''Legend'''

	# hormone binding		# hormone binding
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	# modulation by cellular signalling pathways		# modulation by cellular signalling pathways
	# antagonist resistance		# antagonist resistance
			\|}

S8600021 at 20:47, 25 September 2011

2011-09-25T20:47:57Z

← Older revision		Revision as of 07:47, 26 September 2011
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	Original file name: Figure 2 Big_nrs05003.f2.gif http://www.nursa.org/ejournal/published/05003/big_nrs05003.f2.gif		Original file name: Figure 2 Big_nrs05003.f2.gif http://www.nursa.org/ejournal/published/05003/big_nrs05003.f2.gif
			===Reference===

	=~~=Reference==~~		<pubmed>17464358</pubmed>\| [http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1853070 PMC1853070] \| [http://www.nursa.org/article.cfm?doi=10.1621/nrs.05003 Nucl Recept Signal.]

	~~<pubmed>17464358</pubmed>\| [http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1853070 PMC1853070] \| [http://www.nursa.org/article.cfm?doi=10.1621/nrs.05003 Nucl Recept Signal.]~~

	Copyright © 2007, Griekspoor et al. This is an open-access article distributed under the terms of the Creative Commons Non-Commercial Attribution License, which permits unrestricted non-commercial use distribution and reproduction in any medium, provided the original work is properly cited.		Copyright © 2007, Griekspoor et al. This is an open-access article distributed under the terms of the Creative Commons Non-Commercial Attribution License, which permits unrestricted non-commercial use distribution and reproduction in any medium, provided the original work is properly cited.

	[[Category:Molecular]] [[Category:Endocrine]]		[[Category:Molecular]] [[Category:Endocrine]] [[Category:Cartoon]]

S8600021: /* Steroid hormone receptor signaling */

2010-10-05T08:15:51Z

Steroid hormone receptor signaling

@@ Line 1: / Line 1: @@
 ==Steroid hormone receptor signaling==
-Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors. Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock proteins, and translocates to the nucleus. There, the receptor dimerizes, binds specific sequences in the DNA, called Hormone Responsive Elements or HREs, and recruits a number of coregulators that facilitate gene transcription. This latter step can be modulated by receptor antagonists like tamoxifen (T), and cellular signalling pathways. Examples of processes studied using biophysical techniques and discussed in this review include: hormone binding (1), chaperone interaction (2), nuclear translocation (3), receptor dimerization (4), DNA binding (5), putative membrane-bound receptors (6), coregulator recruitment (7), transcription (8), proteasomal degradation (9), modulation by cellular signalling pathways (10), and antagonist resistance (11).
+Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors. Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock proteins, and translocates to the nucleus. There, the receptor dimerizes, binds specific sequences in the DNA, called Hormone Responsive Elements or HREs, and recruits a number of coregulators that facilitate gene transcription. This latter step can be modulated by receptor antagonists like tamoxifen (T), and cellular signalling pathways.
 Original file name: Figure 2 Big_nrs05003.f2.gif http://www.nursa.org/ejournal/published/05003/big_nrs05003.f2.gif

S8600021: ==Steroid hormone receptor signaling== Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors. Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock

2010-10-05T08:11:54Z

New page

==Steroid hormone receptor signaling==
Steroid Hormone Receptors (SHR) act as hormone dependent nuclear transcription factors. Upon entering the cell by passive diffusion, the hormone (H) binds the receptor, which is subsequently released from heat shock proteins, and translocates to the nucleus. There, the receptor dimerizes, binds specific sequences in the DNA, called Hormone Responsive Elements or HREs, and recruits a number of coregulators that facilitate gene transcription. This latter step can be modulated by receptor antagonists like tamoxifen (T), and cellular signalling pathways. Examples of processes studied using biophysical techniques and discussed in this review include: hormone binding (1), chaperone interaction (2), nuclear translocation (3), receptor dimerization (4), DNA binding (5), putative membrane-bound receptors (6), coregulator recruitment (7), transcription (8), proteasomal degradation (9), modulation by cellular signalling pathways (10), and antagonist resistance (11).

Original file name: Figure 2 Big_nrs05003.f2.gif http://www.nursa.org/ejournal/published/05003/big_nrs05003.f2.gif

==Reference==

<pubmed>17464358</pubmed>| [http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1853070 PMC1853070] | [http://www.nursa.org/article.cfm?doi=10.1621/nrs.05003 Nucl Recept Signal.]

Copyright © 2007, Griekspoor et al. This is an open-access article distributed under the terms of the Creative Commons Non-Commercial Attribution License, which permits unrestricted non-commercial use distribution and reproduction in any medium, provided the original work is properly cited.

[[Category:Molecular]] [[Category:Endocrine]]