Cardiovascular System - Ventricular Septal Defects - Revision history

Z8600021 at 00:58, 14 November 2019

2019-11-14T00:58:26Z

← Older revision		Revision as of 10:58, 14 November 2019
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			* '''Australia - Congenital heart disease hospitalisations, principal diagnosis, by condition and sex, 2016–17'''<ref>Australian Institute of Health and Welfare 2019. Congenital heart disease in Australia. Cat. no. [https://www.aihw.gov.au/reports/heart-stroke-vascular-diseases/congenital-heart-disease-in-australia/contents/table-of-contents CDK 14]. Canberra: AIHW.</ref> "In 2016–17, there were around 4,900 hospitalisations in Australia where congenital heart disease was the principal diagnosis—a rate of 20 hospitalisations per 100,000 population. The highest rate of hospitalisation for a specific form of congenital heart disease was for {{atrial septal defects}} (6.6 hospitalisations per 100,000 population), followed by {{ventricular septal defect}} (1.8), {{transposition of the great vessels}} (1.2), {{patent ductus arteriosus‎}} and {{coarctation of the aorta}}."

	* '''A new anatomic approach of the ventricular septal defect in the interruption of the aortic arch'''{{#pmid:30525196\|PMID30525196}} "The aim of this study was to analyse the anatomy of the ventricular septal defect (VSD) in heart specimens with interruption of the aortic arch (IAA) in order to explore the hypothesis of different embryologic mechanisms for the different anatomic types of IAA. We examined 42 human heart specimens, 25 with IAA as the main disease with concordant atrioventricular and ventriculo-arterial connections and two distinct great arteries, and 17 hearts with IAA associated with other malformations [six common arterial trunk (CAT), five double-outlet right ventricle (DORV), three transposition of the great arteries (TGA), three atrioventricular septal defect (AVSD)]. The interruption was classified according to Celoria and Patton. ...These results reinforce the hypothesis that different pathogenic mechanisms are responsible for the two types of IAA, and the inclusion of IAA type B in the group of neural crest defects. Conversely, IAA type A could be due to overlapping mechanisms: flow-related defect (coarctation-like) and neural crest contribution."		* '''A new anatomic approach of the ventricular septal defect in the interruption of the aortic arch'''{{#pmid:30525196\|PMID30525196}} "The aim of this study was to analyse the anatomy of the ventricular septal defect (VSD) in heart specimens with interruption of the aortic arch (IAA) in order to explore the hypothesis of different embryologic mechanisms for the different anatomic types of IAA. We examined 42 human heart specimens, 25 with IAA as the main disease with concordant atrioventricular and ventriculo-arterial connections and two distinct great arteries, and 17 hearts with IAA associated with other malformations [six common arterial trunk (CAT), five double-outlet right ventricle (DORV), three transposition of the great arteries (TGA), three atrioventricular septal defect (AVSD)]. The interruption was classified according to Celoria and Patton. ...These results reinforce the hypothesis that different pathogenic mechanisms are responsible for the two types of IAA, and the inclusion of IAA type B in the group of neural crest defects. Conversely, IAA type A could be due to overlapping mechanisms: flow-related defect (coarctation-like) and neural crest contribution."

Z8600021 at 00:55, 14 November 2019

2019-11-14T00:55:18Z

← Older revision		Revision as of 10:55, 14 November 2019
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	==Some Recent Findings==		==Some Recent Findings==
			[[File:Australia Congenital heart disease 2016–17.png\|thumb\|alt=Australia Congenital heart disease 2016–17\|Australia Congenital heart disease 2016–17]]
	{\|		{\|
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Z8600021 at 00:53, 14 November 2019

2019-11-14T00:53:49Z

← Older revision		Revision as of 10:53, 14 November 2019
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	[[File:Ventricular_Septal_Defect.jpg\|thumb\|300px\|Ventricular Septal Defect]]		[[File:Ventricular_Septal_Defect.jpg\|thumb\|300px\|Ventricular Septal Defect]]

	The {{Ventricular septal defect}} (VSD) is the ~~most~~ common ~~form~~ of congenital cardiovascular anomaly, occurring in nearly 50% of all infants with a congenital heart defect. Usually occurs in the membranous (perimembranous) rather than muscular interventricular septum, and is more frequent in males that females. This defect can also contribute to outflow tract (OFT) malformations. See also the recent cardiac development online review section - [[Paper_-_Teratogenecity_in_the_setting_of_cardiac_development_and_maldevelopment#Normal_as_opposed_to_Abnormal_Ventricular_Septation\|Normal as opposed to abnormal ventricular septation]].		The {{Ventricular septal defect}} (VSD) is one of the common forms of congenital cardiovascular anomaly, occurring in nearly 50% of all infants with a congenital heart defect. Usually occurs in the membranous (perimembranous) rather than muscular interventricular septum, and is more frequent in males that females. This defect can also contribute to outflow tract (OFT) malformations. See also the recent cardiac development online review section - [[Paper_-_Teratogenecity_in_the_setting_of_cardiac_development_and_maldevelopment#Normal_as_opposed_to_Abnormal_Ventricular_Septation\|Normal as opposed to abnormal ventricular septation]].

	Perimembranous defects are located close to the aortic and tricuspid valves and adjacent to atrioventricular conduction bundle.		Perimembranous defects are located close to the aortic and tricuspid valves and adjacent to atrioventricular conduction bundle.

Z8600021 at 06:06, 3 July 2019

2019-07-03T06:06:55Z

← Older revision		Revision as of 16:06, 3 July 2019
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	[[File:Ventricular_Septal_Defect.jpg\|thumb\|300px\|Ventricular Septal Defect]]		[[File:Ventricular_Septal_Defect.jpg\|thumb\|300px\|Ventricular Septal Defect]]

	The Ventricular ~~Septal Defect~~ (VSD) is the most common form of congenital cardiovascular anomaly, occurring in nearly 50% of all infants with a congenital heart defect. Usually occurs in the membranous (perimembranous) rather than muscular interventricular septum, and is more frequent in males that females. This defect can also contribute to outflow tract (OFT) malformations. See also the recent cardiac development online review section - [[Paper_-_Teratogenecity_in_the_setting_of_cardiac_development_and_maldevelopment#Normal_as_opposed_to_Abnormal_Ventricular_Septation\|Normal as opposed to abnormal ventricular septation]].		The {{Ventricular septal defect}} (VSD) is the most common form of congenital cardiovascular anomaly, occurring in nearly 50% of all infants with a congenital heart defect. Usually occurs in the membranous (perimembranous) rather than muscular interventricular septum, and is more frequent in males that females. This defect can also contribute to outflow tract (OFT) malformations. See also the recent cardiac development online review section - [[Paper_-_Teratogenecity_in_the_setting_of_cardiac_development_and_maldevelopment#Normal_as_opposed_to_Abnormal_Ventricular_Septation\|Normal as opposed to abnormal ventricular septation]].

	Perimembranous defects are located close to the aortic and tricuspid valves and adjacent to atrioventricular conduction bundle.		Perimembranous defects are located close to the aortic and tricuspid valves and adjacent to atrioventricular conduction bundle.
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	{{Heart Abnormal}}		{{Heart Abnormal}}
			<br>
	{{Heart Links}}		{{Heart Links}}

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	* '''Tricuspid Valve Detachment in Ventricular Septal Defect Closure Does Not Impact Valve Function'''{{#pmid:29625102\|PMID29625102}} "Although tricuspid valve detachment (TVD) facilitates improved exposure during trans-atrial ventricular septal defect (VSD) closure, few have analyzed the impact of TVD on long-term valve durability. RESULTS: 247 patients underwent VSD closure; 164 (66.4%) without TVD and 83 (33.6%) with TVD. Median follow-up time was 2343 days (IQR:1237-3963.5) in the group without TVD versus 1606 days (IQR:826-3017) in those with TVD. After successfully matching 83 patients, 29/83 (34.9%) in the non-TVD group had mild TR versus 28/83 (33.7%) in the TVD group (p=0.87). 2 patients in the non-TVD group had moderate TR versus 1 patient in the TVD group at long term follow up. 1 patient in each group suffered transient AV block, but neither required pacemaker insertion. CONCLUSIONS: Tricuspid valve detachment did not compromise long-term valve durability and did not impose increased morbidity. Patients who underwent TVD had a similar prevalence of mild TR to patients without TVD. Moderate TR was exceptionally rare in both groups. When exposure is difficult, TVD is a safe and effective technical adjunct."		* '''Tricuspid Valve Detachment in Ventricular Septal Defect Closure Does Not Impact Valve Function'''{{#pmid:29625102\|PMID29625102}} "Although tricuspid valve detachment (TVD) facilitates improved exposure during trans-atrial ventricular septal defect (VSD) closure, few have analyzed the impact of TVD on long-term valve durability. RESULTS: 247 patients underwent VSD closure; 164 (66.4%) without TVD and 83 (33.6%) with TVD. Median follow-up time was 2343 days (IQR:1237-3963.5) in the group without TVD versus 1606 days (IQR:826-3017) in those with TVD. After successfully matching 83 patients, 29/83 (34.9%) in the non-TVD group had mild TR versus 28/83 (33.7%) in the TVD group (p=0.87). 2 patients in the non-TVD group had moderate TR versus 1 patient in the TVD group at long term follow up. 1 patient in each group suffered transient AV block, but neither required pacemaker insertion. CONCLUSIONS: Tricuspid valve detachment did not compromise long-term valve durability and did not impose increased morbidity. Patients who underwent TVD had a similar prevalence of mild TR to patients without TVD. Moderate TR was exceptionally rare in both groups. When exposure is difficult, TVD is a safe and effective technical adjunct."

	* '''Complete Repair of Coarctation of the Aorta and a Ventricular Septal Defect in a 1,480 g Low Birth Weight Neonate'''{{#pmid:22263148\|PMID22263148}} "Although outcomes of neonatal cardiac surgery have dramatically improved in the last two decades, low body weight still constitutes an important risk factor for morbidity and mortality. In particular, cardiac surgery in neonates with very low birth weight (≤1.5 kg) is carried out with greater risk because most organ systems are immature. We report here on a successful case of early one-stage total repair of coarctation of the aorta and a ventricular septal defect in a 1,480 gram neonate."
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			{\| class="wikitable mw-collapsible mw-collapsed"
			! Older papers
			\|-
			\| {{Older papers}}
			* '''Complete Repair of Coarctation of the Aorta and a Ventricular Septal Defect in a 1,480 g Low Birth Weight Neonate'''{{#pmid:22263148\|PMID22263148}} "Although outcomes of neonatal cardiac surgery have dramatically improved in the last two decades, low body weight still constitutes an important risk factor for morbidity and mortality. In particular, cardiac surgery in neonates with very low birth weight (≤1.5 kg) is carried out with greater risk because most organ systems are immature. We report here on a successful case of early one-stage total repair of coarctation of the aorta and a ventricular septal defect in a 1,480 gram neonate."
			\|}
	==Anatomy==		==Anatomy==
	{\|		{\|

Z8600021 at 03:13, 16 Haziran 2019

2019-06-16T03:13:26Z

← Older revision		Revision as of 13:13, 16 Haziran 2019
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	\| [[File:Mark_Hill.jpg\|90px\|left]] {{Most_Recent_Refs}}		\| [[File:Mark_Hill.jpg\|90px\|left]] {{Most_Recent_Refs}}

	Search term: [http://www.ncbi.nlm.nih.gov/pubmed/?term=Ventricular+Septal+Defect ''Ventricular Septal Defect~~''] \| [http://www.ncbi.nlm.nih.gov/pubmed/?term=Fontan+procedure ''Fontan procedure~~'']		Search term: [http://www.ncbi.nlm.nih.gov/pubmed/?term=Ventricular+Septal+Defect ''Ventricular Septal Defect'']

	\|}		\|}
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	# '''Grade V''' - complex plexiform, angiomatous and cavernous lesions and hyalinization of intimal fibrosis.		# '''Grade V''' - complex plexiform, angiomatous and cavernous lesions and hyalinization of intimal fibrosis.
	# '''Grade VI''' - necrotizing arteritis.		# '''Grade VI''' - necrotizing arteritis.

	~~==Fontan Procedure==~~

	~~The Fontan procedure or Fontan–Kreutzer procedure is a surgical procedure used in children with univentricular hearts.~~

	==Movies==		==Movies==

Z8600021 at 03:12, 16 Haziran 2019

2019-06-16T03:12:03Z

← Older revision		Revision as of 13:12, 16 Haziran 2019
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	\| [[File:Mark_Hill.jpg\|90px\|left]] {{Most_Recent_Refs}}		\| [[File:Mark_Hill.jpg\|90px\|left]] {{Most_Recent_Refs}}

	Search term: [http://www.ncbi.nlm.nih.gov/pubmed/?term=Ventricular+Septal+Defect ''Ventricular Septal Defect'']		Search term: [http://www.ncbi.nlm.nih.gov/pubmed/?term=Ventricular+Septal+Defect ''Ventricular Septal Defect''] \| [http://www.ncbi.nlm.nih.gov/pubmed/?term=Fontan+procedure ''Fontan procedure'']

	\|}		\|}
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	# '''Grade V''' - complex plexiform, angiomatous and cavernous lesions and hyalinization of intimal fibrosis.		# '''Grade V''' - complex plexiform, angiomatous and cavernous lesions and hyalinization of intimal fibrosis.
	# '''Grade VI''' - necrotizing arteritis.		# '''Grade VI''' - necrotizing arteritis.

			==Fontan Procedure==

			The Fontan procedure or Fontan–Kreutzer procedure is a surgical procedure used in children with univentricular hearts.

	==Movies==		==Movies==

Z8600021 at 23:39, 10 March 2019

2019-03-10T23:39:10Z

← Older revision		Revision as of 09:39, 11 March 2019
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			* '''A new anatomic approach of the ventricular septal defect in the interruption of the aortic arch'''{{#pmid:30525196\|PMID30525196}} "The aim of this study was to analyse the anatomy of the ventricular septal defect (VSD) in heart specimens with interruption of the aortic arch (IAA) in order to explore the hypothesis of different embryologic mechanisms for the different anatomic types of IAA. We examined 42 human heart specimens, 25 with IAA as the main disease with concordant atrioventricular and ventriculo-arterial connections and two distinct great arteries, and 17 hearts with IAA associated with other malformations [six common arterial trunk (CAT), five double-outlet right ventricle (DORV), three transposition of the great arteries (TGA), three atrioventricular septal defect (AVSD)]. The interruption was classified according to Celoria and Patton. ...These results reinforce the hypothesis that different pathogenic mechanisms are responsible for the two types of IAA, and the inclusion of IAA type B in the group of neural crest defects. Conversely, IAA type A could be due to overlapping mechanisms: flow-related defect (coarctation-like) and neural crest contribution."

	* '''Tricuspid Valve Detachment in Ventricular Septal Defect Closure Does Not Impact Valve Function'''{{#pmid:29625102\|PMID29625102}} "Although tricuspid valve detachment (TVD) facilitates improved exposure during trans-atrial ventricular septal defect (VSD) closure, few have analyzed the impact of TVD on long-term valve durability. RESULTS: 247 patients underwent VSD closure; 164 (66.4%) without TVD and 83 (33.6%) with TVD. Median follow-up time was 2343 days (IQR:1237-3963.5) in the group without TVD versus 1606 days (IQR:826-3017) in those with TVD. After successfully matching 83 patients, 29/83 (34.9%) in the non-TVD group had mild TR versus 28/83 (33.7%) in the TVD group (p=0.87). 2 patients in the non-TVD group had moderate TR versus 1 patient in the TVD group at long term follow up. 1 patient in each group suffered transient AV block, but neither required pacemaker insertion. CONCLUSIONS: Tricuspid valve detachment did not compromise long-term valve durability and did not impose increased morbidity. Patients who underwent TVD had a similar prevalence of mild TR to patients without TVD. Moderate TR was exceptionally rare in both groups. When exposure is difficult, TVD is a safe and effective technical adjunct."		* '''Tricuspid Valve Detachment in Ventricular Septal Defect Closure Does Not Impact Valve Function'''{{#pmid:29625102\|PMID29625102}} "Although tricuspid valve detachment (TVD) facilitates improved exposure during trans-atrial ventricular septal defect (VSD) closure, few have analyzed the impact of TVD on long-term valve durability. RESULTS: 247 patients underwent VSD closure; 164 (66.4%) without TVD and 83 (33.6%) with TVD. Median follow-up time was 2343 days (IQR:1237-3963.5) in the group without TVD versus 1606 days (IQR:826-3017) in those with TVD. After successfully matching 83 patients, 29/83 (34.9%) in the non-TVD group had mild TR versus 28/83 (33.7%) in the TVD group (p=0.87). 2 patients in the non-TVD group had moderate TR versus 1 patient in the TVD group at long term follow up. 1 patient in each group suffered transient AV block, but neither required pacemaker insertion. CONCLUSIONS: Tricuspid valve detachment did not compromise long-term valve durability and did not impose increased morbidity. Patients who underwent TVD had a similar prevalence of mild TR to patients without TVD. Moderate TR was exceptionally rare in both groups. When exposure is difficult, TVD is a safe and effective technical adjunct."

Z8600021 at 23:10, 10 March 2019

2019-03-10T23:10:16Z

← Older revision		Revision as of 09:10, 11 March 2019
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	! {{ICD-11}}		! {{ICD-11}}
	\|-bgcolor="FEF9E7"		\|-bgcolor="FEF9E7"
	\| ~~[https://icd.who.int/browse11/l-m/en#/http://id.who.int/icd/entity/~~668140715 '''LA88.4''' Ventricular septal defect]		\| {{ICD11weblink}}668140715 '''LA88.4''' Ventricular septal defect]
	:''A congenital cardiac malformation in which there is a hole or pathway between the ventricular chambers.''		:''A congenital cardiac malformation in which there is a hole or pathway between the ventricular chambers.''
	~~[https://icd.who.int/browse11/l-m/en#/http://id.who.int/icd/entity/~~658059528 LA88.40 Trabecular muscular ventricular septal defect] \| ~~[https://icd.who.int/browse11/l-m/en#/http://id.who.int/icd/entity/~~2023258628 LA88.41 Perimembranous central ventricular septal defect] \| ~~[https://icd.who.int/browse11/l-m/en#/http://id.who.int/icd/entity/~~924554858 LA88.42 Ventricular septal defect haemodynamically insignificant]
			{{ICD11weblink}}658059528 LA88.40 Trabecular muscular ventricular septal defect] \| {{ICD11weblink}}2023258628 LA88.41 Perimembranous central ventricular septal defect] \| {{ICD11weblink}}924554858 LA88.42 Ventricular septal defect haemodynamically insignificant]
	\|}		\|}
	{{ICD-11-Circulatory system structural anomalies table}}		{{ICD-11-Circulatory system structural anomalies table}}

Z8600021 at 04:23, 8 March 2019

2019-03-08T04:23:46Z

@@ Line 1: / Line 1: @@
 {{Header}}
 =LA88.4 Ventricular Septal Defect=
 {|
 |-bgcolor="FFCC00"
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 Search term: [http://www.ncbi.nlm.nih.gov/pubmed/?term=Ventricular+Septal+Defect ''Ventricular Septal Defect'']
 |}
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 # '''Grade V''' - complex plexiform, angiomatous and cavernous lesions and hyalinization of intimal fibrosis.
 # '''Grade VI''' - necrotizing arteritis.
 ==Movies==

Z8600021 at 04:22, 8 March 2019

2019-03-08T04:22:39Z

← Older revision		Revision as of 14:22, 8 March 2019
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	{{Header}}		{{Header}}
	=~~LB21~~.~~5 Congenital~~ Ventricular Septal ~~Defects~~=		=LA88.4 Ventricular Septal Defect=
	ICD-11 Beta Draft code: LB21.5. Current ICD-10 code: [[Cardiovascular_System_-_Ventricular_Septal_Defects#Congenital_malformations_of_the_circulatory_system_.28Q20-Q28.29\|Q21.0 Ventricular septal defect]]

			{\|
			\|-bgcolor="FFCC00"
			! {{ICD-11}}
			\|-bgcolor="FEF9E7"
			\| [https://icd.who.int/browse11/l-m/en#/http://id.who.int/icd/entity/668140715 '''LA88.4''' Ventricular septal defect]
			:''A congenital cardiac malformation in which there is a hole or pathway between the ventricular chambers.''
			[https://icd.who.int/browse11/l-m/en#/http://id.who.int/icd/entity/658059528 LA88.40 Trabecular muscular ventricular septal defect] \| [https://icd.who.int/browse11/l-m/en#/http://id.who.int/icd/entity/2023258628 LA88.41 Perimembranous central ventricular septal defect] \| [https://icd.who.int/browse11/l-m/en#/http://id.who.int/icd/entity/924554858 LA88.42 Ventricular septal defect haemodynamically insignificant]
			\|}
	{{ICD-11-Circulatory system structural anomalies table}}		{{ICD-11-Circulatory system structural anomalies table}}
	==Introduction==		==Introduction==